H. DALDAL And M. NAZIROĞLU, "Carvacrol protects the ARPE19 retinal pigment epithelial cells against high glucose-induced oxidative stress, apoptosis, and inflammation by suppressing the TRPM2 channel signaling pathways," Graefe's Archive for Clinical and Experimental Ophthalmology , vol.260, no.8, pp.2567-2583, 2022
DALDAL, H. And NAZIROĞLU, M. 2022. Carvacrol protects the ARPE19 retinal pigment epithelial cells against high glucose-induced oxidative stress, apoptosis, and inflammation by suppressing the TRPM2 channel signaling pathways. Graefe's Archive for Clinical and Experimental Ophthalmology , vol.260, no.8 , 2567-2583.
DALDAL, H., & NAZIROĞLU, M., (2022). Carvacrol protects the ARPE19 retinal pigment epithelial cells against high glucose-induced oxidative stress, apoptosis, and inflammation by suppressing the TRPM2 channel signaling pathways. Graefe's Archive for Clinical and Experimental Ophthalmology , vol.260, no.8, 2567-2583.
DALDAL, HATİCE, And Mustafa NAZIROĞLU. "Carvacrol protects the ARPE19 retinal pigment epithelial cells against high glucose-induced oxidative stress, apoptosis, and inflammation by suppressing the TRPM2 channel signaling pathways," Graefe's Archive for Clinical and Experimental Ophthalmology , vol.260, no.8, 2567-2583, 2022
DALDAL, HATİCE And NAZIROĞLU, Mustafa. "Carvacrol protects the ARPE19 retinal pigment epithelial cells against high glucose-induced oxidative stress, apoptosis, and inflammation by suppressing the TRPM2 channel signaling pathways." Graefe's Archive for Clinical and Experimental Ophthalmology , vol.260, no.8, pp.2567-2583, 2022
DALDAL, H. And NAZIROĞLU, M. (2022) . "Carvacrol protects the ARPE19 retinal pigment epithelial cells against high glucose-induced oxidative stress, apoptosis, and inflammation by suppressing the TRPM2 channel signaling pathways." Graefe's Archive for Clinical and Experimental Ophthalmology , vol.260, no.8, pp.2567-2583.
@article{article, author={HATİCE DALDAL And author={Mustafa NAZIROĞLU}, title={Carvacrol protects the ARPE19 retinal pigment epithelial cells against high glucose-induced oxidative stress, apoptosis, and inflammation by suppressing the TRPM2 channel signaling pathways}, journal={Graefe's Archive for Clinical and Experimental Ophthalmology}, year=2022, pages={2567-2583} }